AHAD Lecture 4

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  • AHAD - Lecture 4 (cows)
    • Mastitis
      • inflammatoion of mammary glands
      • reduces milk prodoction, fertility and increases risk of early culling. this is costly
      • more prevalent in the dairy industry
      • appearance
        • subclinical
          • things seem normal but there is some inflammation of the udder which needs investigating
            • need to test milk, this can either be done farmside or in a lab
              • on the farm, california milk test is carried out. Milk is mixed with reagent, reaction will occur and turn gelatenous and lumpy if there is high SCC
              • lab will test SCC count, and compare to threshold
                • > 200,000 SCC is threshold for subclinical mastitis
        • clinical
          • external signs of mastitis that do not require further examining.
            • milk: changes in colour, composition, output and somatic cell count (SCC) (1)
              • further, does it smell, is it clotted, stringy, thick?
            • udder: hard, hot, tender to touch, skin may redden (1,2)
              • could be gangrenous caused by staphylococcus aureus (3)
            • cow: changes in behavour (1,2,3,)
              • decreased food intake, recumbent, collapsed, increased heart and respiratory rate
          • 3 different gradings, 1 (mild), 2 (moderate), 3 (severe)
      • aetiology
        • contagious pathogens found on cows
          • staphylococcus aureus
          • streptococcus agalactiae
          • streptococcus dysgalactiae
          • found in natural flora on udder
          • infected cows are primary reservoir so must be segregted and milked last
          • 1-2
            • 3 different gradings, 1 (mild), 2 (moderate), 3 (severe)
          • 5 point plan to prevent this: 1) milking machine maintenance 2) post-milk teat disinfection   3) dry cow           therapy      4) treat clinical cases           5) cull chronic cows
      • duration
        • acute
          • sudden and over a short duration, often clinical
          • cows will be unwell and can even end in dealth
        • chronic
          • persists for a long duration, clinical or subclinical
          • cows usually look well
          • clinical signs are mild but can end in permanent udder damage
      • udder defences
        • anatomical defence - barrier for protection
        • innate immune defence - immune response if bacteria entry occurs
      • treated with antibiotics
    • Metabolic diseases
      • associated with high production systems
      • hypomagnesaemia = grass staggers
        • common in beef suckler cows, young growing grass has less magnesium so causes deficiencies
        • neurological signs: twitching, abnormal behaviours, odd urination, seizures, or sudden death
      • hypocalcaemia = milk fever
        • jerseys and gurnseys are predisposed to this
        • commin in high yielding or older cows
        • weak and recumbent, weak neck muscles, called swan neck
      • ketosis
        • fat is mobilised into energy, often seen with cows who are lactating or in a NEG
        • vague clinical sign, if not treated will lead to death, tho luckily we know they are susceptible during pregnancy / lactation so on the look out for signs
          • measure ketones in blood, milk, urine to ensure correct diagnosis
        • treated with propylene glycol orally
          • prevent by optimising nutriton before and during lactation and do not feed buturic silage (often in poor quality silage)
    • Lameness
      • abnormal gate or posture that results from pain in the limbs
        • though can also occur through neurological conditions which aren't painful
      • results in: reduced milk yield, reduced fertility, vet costs, culling costs, treatment costs, labour costs
      • 0-3 scoring system, looks at weight bearing, rhythm, fluid strides, back (flat or arched)
      • most cases in hoof
        • sole ulcers = non-infectious
        • white line disease = non-infectious
        • digital dermititis = infectious

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