at G2/M, cap-dependant translation is inhibited. elF4E phosphorylation is reduced, and there is increase phosphorylation of elf2A and eEF2.
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Is all translation stopped at G2/M?
No- specific genes with IRES sequences are still translated
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Example of viruses regulating translation
picornaviruses - eg, poliovirus and encephalomyocarditis, cleave elf4g and PAMP
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How does viral mRNA escape inhibition?
they all have internal ribosomal entry sites.
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How do IRES control translation?
forms stem loop structures that allow translation even when cap dependant initation is blocked
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IRE-IRP interactions when the iron concentration is high
When iron concentration is HIGH - it binds IRP, preventing IRE binding, allowing translation of ferritin (storage) but decreasing transferrin receptors.
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